Claes B. and an increased nuclear pool of Ccnd2 via Prlr and Jak2. Consistent with these results, the loss of MafA resulted in impaired proliferation of -cells at 4 weeks of age. These results suggest that MafA regulates the postnatal proliferation of -cells via prolactin signaling. Introduction Accumulating evidence suggests that postnatal organ development and … Continue reading Claes B